Trigeminal Neuralgia
Summary
- Chronic neuropathic pain disorder affecting the trigeminal nerve
- Characterised by sudden, severe, electric shock-like pain in the face
- Diagnosis primarily clinical, with MRI to rule out secondary causes
Pathophysiology
- Neurovascular compression of the trigeminal nerve root entry zone, most commonly by superior cerebellar artery
- Demyelination of trigeminal sensory fibres leading to ephaptic transmission
- Central sensitization and hyperexcitability of trigeminal nuclei
Demographics
- Incidence: 4-13 per 100,000 person-years
- Peak onset: 50-60 years of age
- Female to male ratio: 1.5-2:1
- Higher prevalence in patients with multiple sclerosis (1-6.3%)
Diagnosis
- Based on clinical presentation and patient history
- International Headache Society diagnostic criteria:
- At least three attacks of unilateral facial pain
- Occurring in one or more divisions of the trigeminal nerve
- Pain has at least three of the following characteristics:
- Recurring in paroxysmal attacks
- Severe intensity
- Electric shock-like, shooting, stabbing, or sharp in quality
- Lasting from a fraction of a second to 2 minutes
- Triggered by innocuous stimuli
- No clinically evident neurological deficit
Imaging
- MRI brain with contrast:
- To exclude secondary causes (e.g., tumours, multiple sclerosis)
- High-resolution T2-weighted sequences to visualise neurovascular compression
- MR angiography:
- To evaluate vascular anatomy and identify compressing vessels
- DTI and tractography:
- To assess microstructural changes in trigeminal nerve
Treatment
- Medical management:
- First-line: Carbamazepine or oxcarbazepine
- Second-line: Gabapentin, pregabalin, or baclofen
- Third-line: Lamotrigine or phenytoin
- Surgical interventions:
- Microvascular decompression
- Stereotactic radiosurgery (Gamma Knife)
- Percutaneous procedures:
- Glycerol rhizotomy
- Radiofrequency thermocoagulation
- Balloon compression
- Neuromodulation:
- Peripheral nerve stimulation
- Motor cortex stimulation

